Why nose breathing is important.
Encouraging a normal volume of air to be breathed
Nose breathing usually results in breathing a Minute Volume of air, which is closer to the physiological norm. Mouth breathers tend to breathe too much air for their level of activity. We should normally breathe around 4-5 litres of air per minute at rest. Breathing a greater volume of air per minute at rest tends to lower the End Tidal Carbon Dioxide (ETCO2) level. Essentially this means we carry a smaller % of carbon dioxide (CO2) in our blood and our lungs.
Surprisingly, CO2 is not just a waste gas. It is essential that we retain a certain amount inside our blood in order for the body to work properly.
I’m going to try to explain in a nutshell why there is a huge reason to be concerned about low levels of CO2 in our blood and lungs…. And it is mostly to do with our ability to achieve good cellular oxygenation.
Carbon dioxide - one of nature’s miracle gases
CO2 is a bronchodilator and vasodilator, and as such opens up air passages and blood vessels. This means normalising (increasing) CO2 levels can reduce blood pressure. It’s well documented that breathing dysfunction can be related to cardiovascular problems (1).
In addition, there is the Bohr Effect – so when CO2 is increased (normalised) by breathing a smaller (normal) volume of air, haemoglobin more readily releases oxygen (O2), and thus allows any cells which may be suffering from cellular hypoxia, to become better oxygenated.
Furthermore, we know that altering CO2 levels can help treat the common cold (2), as well as help reduce histamine release from mast cells and so potentially treat hay-fever (3).
It would seem that CO2 may be just as important for health as is O2. It just seems to have gotten a bad press, perhaps due to its links with global warming and dirty power stations.
The symptoms of cellular hypoxia
The symptom of lack of oxygenation will differ according to:
(a): the individual’s genetic predisposition
e.g.: “the asthma gene” leads to a bronchospasm when the smooth muscle which usually keeps the airways open is not oxygenated (in addition to when there is low CO2 due to the asthmatic’s breathing dysfunction).
(b): the type of cell that is suffering
e.g: a nerve cell suffering cellular hypoxia may respond with a symptom of numbness, peripheral neuropathy or tingling. Whereas a brain cell with insufficient oxygen will deliver a symptom of dizziness – just like when you blow balloons up too fast at a party. There’s plenty of O2 in the air you are breathing. It’s just that you are losing too much CO2 into the balloon and so the oxygen is not being effectively released from your haemoglobin (Bohr Effect as above).
(c): other factors
There are many factors beyond the scope of this article, which could affect the symptoms, but factors such as extended periods of time with lack of oxygen (chronic cellular hypoxia as opposed to acute cellular hypoxia) will play a role.
So how can a mouth breather become a nose breather?
The good news is that learning to breathe in a different way than your habitual norm, can improve ETCO2 levels, thus resulting in better cellular oxygenation. Therefore, inflammation may reduce and so chronic soft tissue obstruction, such as a persistent blocked nose, or enlarged tonsils may improve over time. It seems that whilst our breathing patterns can become habitually worse, they can also become habitually better, thanks to “respiratory neuroplasticity” (4).
It appears that the younger we are, the more sensitive we are to alterations in breathing patterns (5).
However, improvements in breathing patterns have been recorded across all age ranges, with many cases of reductions in many different symptoms being achieved during the use of a number of different breathing retraining techniques.
One of the more well known techniques is the Buteyko Institute Method (BIM), which focuses on normalising breathing patterns and has been shown to be effective for asthma and sleep apnoea, as well as a number of other symptoms (6-8).
After a typical BIM (5 day) breathing retraining course, the client is asked to undertake a series of exercises. Around 1 hour a day of practice (split up into 5 or 6 lots of 12 minutes) is required for anything between 1 month and 6 months. It takes time, but over time we can consciously make amazing changes to how we breathe, resulting in a better, more functional subconscious breathing pattern.
During these courses, there is more to do than simply learn to nose breathe. However, nose breathing is a key component and is seen as essential to improving health outcomes using breathing techniques.
Nose breathing also allows warming, filtering and humidifying of the air. In addition, the air has the chance to be mixed with nitric oxide formed in the sinuses when taken through the nose (9). Nitric oxide vasodilates and bronchodilates (opens up blood vessels and airways) as well as having potential other health benefits such as being bacteriocidal.
All in all breathing through the nose is good for you, and breathing through the mouth is definitely not in your best interests. If you suffer from poor sleep, congestion, low energy levels, asthma, snoring, or sleep apnoea, ensure that you contact your GP and consider enrolling on a Buteyko Institute Method course.
However, one of the best things you can do for your body is to encourage nose breathing while you sleep.
Use sleepYstrip® to achieve this and breathe better while you sleep. Visit www.sleepystrip.com to find out more about this great product.
How big a problem is poor sleep?
Poor sleep is one of the biggest health crises of our time, with around 20% of some populations recording poor sleep quality (10) with other studies quoting over 50% of people having poor quality sleep (11). This may be related to modern lifestyles such as over-stimulation with caffeine, cigarette smoking and use of sleep medicine (11).
What methods are available to assist with sleeping problems?
Common approaches to reducing signs and symptoms of snoring and sleep apnoea include:
- Using a Continuous Positive Airway Pressure (CPAP) machine to blow air down the airway
- Using a dental splint (MAS) to help keep the lower jaw forwards to open up the airway
- Uvulopalatopharyngoplasty (soft palate surgery) to tighten the soft palate
- Laser treatment to tighten the soft palate
Whilst all of these treatments can have some benefit, they do not directly help change the habitual breathing pattern. Therefore, whilst sleep quality alone may improve – with resulting health benefits, the day time dysfunctional habitual breathing pattern may not change.
It is my hope that the future of treating sleeping disorders will involve at least in part, a focus on the following:
- attempts to improve day time and night time breathing patterns – using breathing retraining programs such as the BIM (www.buteyko.info) together with lifestyle changes, such as healthy eating habits, and a curfew on screen technology late at night.
- encouraging a closed mouth posture at night – using sleepYstrip® (www.sleepystrip.com).
- combining the above methods with MAS / CPAP / surgery only when the above fails.
So to conclude. Why not consider assisting with normal breathing patterns, instead of simply forcing the airway open using machines and appliances?
When there is an absolute need to hold the airway open (with MAS or CPAP or surgery), because of a lack of time to go through breathing retraining (with BIM), or because of a failure of breathing retraining, then at the very least we should ensure that we have a closed mouth posture, so that we may breathe through the nose all night long.
One of the easiest ways to achieve a closed mouth posture while asleep is to use sleepYstrip®. sleepYstrip® can be used in combination with both MAS and nasal CPAP devices.
1. Han JN, Stegen K, Simkens K, Cauberghs M, Schepers R, Van den Bergh O, Clément J, Van de Woestijne KP, Unsteadiness of breathing in patients with hyperventilation syndrome and anxiety disorders, Eur Respir J 1997; 10: p. 167–176.
2. Raising CO2 can decrease nasal resistance and treat seasonal rhinitis. Casale, 2008 J Allergy Clin Immunol. 121, 105-109
3. Treatment of mast cells with carbon dioxide suppresses degranulation via a novel mechanism involving repression of increased intracellular calcium levels J. W. Strider, C. G. Masterson & P. L. Durham. Center for Biomedical & Life Sciences, Missouri State University, Springfield, MO, USA
4. Respiratory control and respiratory plasticity become habitually maladaptive via long-term facilitation (LTF). Baker et al. 2001 Respiratory plasticity: differential actions of continuous and episodic hypoxia and hypocapnia. Resp Phys, 129, 25-35
5. 5. Marcus et al. Developmental Aspects of the Upper Respiratory Airway – Report from an NHLBI workshop, March 5-6, 2009 Proc Am Thorac Soc, 15, 6
6. Bowler SD, Green A, Mitchell CA, Buteyko breathing techniques in asthma: a blinded randomised controlled trial, Med J of Australia 1998; 169: p. 575-578.
7. Patrick McHugh, Fergus Aitcheson, Bruce Duncan and Frank Houghton Buteyko Breathing Technique for asthma: an effective intervention NZMJ 12 December 2003 V 116 No 1187
8. Zahra Mohamed Hassan, Nermine Mounir Riad, Fatma Hassan Ahmed. Effect of Buteyko breathing technique on patients with bronchial asthma. Egyptian Journal of Chest Diseases and Tuberculosis (2012) 61, 235–241
9. 9. High nitric oxide production in human paranasal sinuses. Lundberg JO, Farkas-Szallasi T, Weitzberg E, Rinder J, Lidholm J, Anggaard A, Hokfelt T, Lundberg JM, Alving K. Nat Med 1995 April; 1 (4): 370-3.
10. Prevalence of poor sleep quality and its relationship with body mass index among teenagers: evidence from Taiwan. Sch Health. 2013 Aug;83(8):582-8.
11. The Epidemiology of Sleep Quality, Sleep Patterns, Consumption of Caffeinated Beverages, and Khat
Use among Ethiopian College Students. Seblewengel Lemma, Sheila V. Patel, Yared A. Tarekegn, Mahlet G. Tadesse, Yemane Berhane Bizu Gelaye,and Michelle A. Williams. Addis Continental Institute of Public Health, Addis Ababa, Ethiopia. Multidisciplinary International Research Training Program, Department of Epidemiology, Harvard School of Public Health, 677 Huntington Avenue, Kresge 501, Boston, MA 02115, USA. Department of Mathematics & Statistics, Georgetown University, Washington, DC 20057, USA